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Plant pathogens secrete a collection of effector proteins. These effectors function to promote colonization, typically by altering host physiology or by modulating host immune responses. In the presence of cognate host immune receptor proteins, however, the same effectors trigger host immunity.
Our research largely focusses on the identification and functional characterisation of effectors from two plant-pathogenic fungi, Venturia inaequalis and Cladosporium fulvum, which cause scab and leaf mould disease of apple and tomato, respectively, as well as from the plant-pathogenic bacterium Pseudomonas syringae pv. actinidiae (Psa), which causes canker disease of kiwifruit.
The aim of this research is to understand how these pathogens promote host colonization or trigger host immunity through the deployment of effectors, with the goal of better informing disease resistance breeding programmes in apple, tomato and kiwifruit.
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Last updated on Tuesday 11 July 2017